80% of people worldwide are below cardioprotective levels of omega-3s.
Recent research supports the importance of EPA and DHA for a variety of cardiovascular outcomes, including reducing the risk of coronary heart disease, myocardial infarction and cardiac death.
A groundbreaking new meta-analysis on the topic was recently published in Mayo Clinic Proceedings.
Read below about the many more reasons to recommend omega-3s for your patient's heart.
-
Reduction in Cardiac Death Risk
Several recent meta analyses have shown that consumption of EPA and DHA reduces the risk of cardiac death by a statistically significant 8% and is particularly effective in patients with cardiovascular risk factors.
-
Reduction In Coronary Heart Disease Risk
A recent study demonstrated that EPA and DHA consumption statistically reduced the risk of coronary heart disease (CHD) events as well as CHD death by 10% and 9% respectively.
-
Blood Pressure Reduction
EPA and DHA have been shown to have a statistically significant reduction in systolic and diastolic blood pressure, a risk factor for coronary heart disease. This is as effective as lifestyle changes, like increasing physical activity, and restricting alcohol or sodium intake.
-
Triglyceride Lowering
A large body of evidence has demonstrated a 20-40% reduction in serum triglycerides with increased intake of EPA and DHA omega-3s.
-
Decreased Heart Rate
There is considerable evidence for the role of omega-3s in reducing heart rate as well as heart rate variability.
-
Healthy Blood Vessels
Omega-3s support healthy blood vessels by decreasing arterial stiffness and increasing endothelial function.
-
Myocardial Infarction
Recent research shows that omega-3s statistically reduce the risk of myocardial infarction (MI) by 13% and the risk of fatal MI by 35%.
-
Recovery Post-MI
Emerging research shows that omega-3s have the potential to improve heart function and support recovery following a heart attack.
-
Reduction in Post-Operative Atrial Fibrillation
While the role of omega-3s in the prevention or treatment of atrial fibrillation is not yet fully established, there does appear to be strong evidence suggesting a role in reducing post-operative atrial fibrillation.
